7 research outputs found
Learning styles: Individualizing computerâbased learning environments
In spite of its importance, learning style is a factor that has been largely ignored in the design of educational software. Two issues concerning a specific set of learning styles, described by Honey and Mumford (1986), are considered here. The first relates to measurement and validity. This is discussed in the context of a longitudinal study to test the predictive validity of the questionnaire items against various measures of academic performance, such as course choice and level of attainment in different subjects. The second issue looks at how the learning styles can be used in computerâbased learning environments. A reâexamination of the four learning styles (Activist, Pragmatist, Reflector and Theorist) suggests that they can usefully be characterized using two orthogonal dimensions. Using a limited number of pedagogical building blocks, this characterization has allowed the development of a teaching strategy suitable for each of the learning styles. Further work is discussed, which will use a multiâstrategy basic algebra tutor to assess the effect of matching teaching strategy to learning style
Measuring Servitization Progress and Outcome:The Case of âAdvanced Servicesâ
The purpose of this paper is to establish a framework for assessing the progress and outcome of a manufacturerâs transformation towards becoming a provider of âadvanced servicesâ â a complex bundling of products and services, whereby manufacturers offer capabilities and outcomes instead of products alone. âAdvanced servicesâ represent the most complex offering in the current servitization trend among manufacturers. However, current performance measures lack the breadth and focus to assess progress or outcomes, and so support research and practice of organisational transformation efforts required. To address this gap the paper investigates how a manufacturerâs efforts to become an âadvanced servicesâ provider can be comprehensively measured, and develops a framework for assessing the transformation journey towards becoming an âadvanced servicesâ provider. The research method is based on (1) a systematic literature review process to create a comprehensive set of service-related performance measures that are available to assess a manufacturerâs servitization efforts, followed by (2) an engagement with an expert panel to synthesise the identified measures and create a set of âadvanced servicesâ performance measures. The proposed framework is presented as a scorecard that can be used in practice to assess the progress and outcome of a manufacturerâs transformation towards becoming a provider of âadvanced servicesâ
Juvenile Pagetâs disease with compound heterozygous mutations in TNFRSF11B presenting with recurrent clavicular fractures and a mild skeletal phenotype
Juvenile Pagetâs disease (JPD) is a rare recessively-inherited bone dysplasia. The great majority of cases described to date have had homozygous mutations in TNFRSF11B, the gene encoding osteoprotegerin. We describe a boy who presented with recurrent clavicular fractures following minor trauma (8 fractures from age 2 to 11). He was of normal height and despite mild lateral bowing of the thighs and anterior bowing of the shins he remained physically active. Abnormal modelling was noted in ribs and humeri on clavicular radiographs, and a skeletal survey at the age of 7 showed generalised diaphyseal expansion of the long bones with thickening of the periosteal and endosteal surfaces of the cortices. On biochemical evaluation, serum alkaline phosphatase was noted to be persistently elevated. The diagnosis of JPD was confirmed by the finding of compound heterozygous mutations in TNFRSF11B: a maternally-inherited Aâ>âG missense mutation at position 1 of the first amino acid codon (previously reported) and a paternally-inherited splice acceptor site mutation in intron 3 at a highly conserved position (not previously reported). Bioinformatics analysis suggested both mutations were disease-causing. Compound heterozygote mutations in TNFRSF11B causing JPD have been previously reported only once â in a boy who also had a relatively mild skeletal phenotype. The milder features may lead to delay in diagnosis and diagnostic confusion with other entities, but the extraskeletal features of JPD may nonetheless develop